Hypo Solvestor TT6 200/100©

 

A Made in DE 1

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Hashimoto’s thyroiditis or chronic lymphocytic thyroiditis is an autoimmune disease in which the thyroid gland is attacked by a variety of cell- and antibody-mediated immune processes. Hypothyroidism, often called underactive thyroid or low thyroid and sometimes hypothyreosis, is a common endocrine disorder in which the thyroid gland does not produce enough thyroid hormone. 

HYPO SOLVESTOR TT6 200 or 100© homeophatic medicine

produced in Germany under § 38, Abs.1, S.3 AMG BRD (Law on medicinal products),

with non therapeutic indication.

OK HS TT6 200©

Hashimoto’s thyroiditis   –   1 original packaging Hypo Solvestor TT6 200,  30 or 100 ml of solution contains: Juglans regia, Ammonium carbonicum, Strychnos ignatia, Selenium metallicum, Origanum vulgare, Primula veris, Ferrum metallicum, Fucus vesiculosus, Zingiber officinale, Strontium carbonicum, Agentum metallicum, Ethanol 43% (m/m).

Hypothyroidism   –   1 original packaging Hypo Solvestor TT6 100,  30 or 100 ml of solution contains: Juglans regia, Ammonium carbonicum, Strychnos ignatia, Selenium metallicum, Primula veris, Ferrum metallicum, Fucus vesiculosus, Zingiber officinale, Ethanol 43% (m/m).

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Hypo Solvestor TT6-100 ©  Hypothyroidism 

Hypo Solvestor TT6-200 ©  Hashimoto’s thyroiditis 

Hypo Solvestor TT6 200© is available as 1-month dose £18.50 and 3-month dose £44.50 (regulatory therapy).

ORDER VIA E-MAIL: info@kasfero.com 

KASFERO NATURAL MEDICINE ®

Hyposolvestor TT6 PUR

Hypo Solvestor TT6 © PUR – directly from the nature – a mixture of herbs and fruits – it is used in the context of regulatory therapy under the supervision of an authorized therapist. Find an authorized therapists here CLUB HP 300

Hypothyroidism (/ˌhpɵˈθaɪərɔɪdɪzəm/), from hypo- (“under” in Greek) and thyroid (the thyroid gland), often called underactive thyroid or low thyroid and sometimes hypothyreosis, is a common endocrine disorder in which the thyroid gland does not produce enough thyroid hormone. It can cause a number of symptoms, such as tiredness, poor ability to tolerate cold, and weight gain. In children, hypothyroidism leads to delays in growth and intellectual development, which is called cretinism in severe cases. The diagnosis of hypothyroidism, when suspected, can be confirmed with blood tests measuring thyroid-stimulating hormone (TSH) and thyroxine levels.

Worldwide, too little iodine in the diet is the most common cause of hypothyroidism. In countries with enough dietary iodine, the most common cause of hypothyroidism is the autoimmune condition Hashimoto’s thyroiditis. Less common causes include the following: previous treatment with radioactive iodine, injury to the hypothalamus or the anterior pituitary gland, certain medications, a lack of a functioning thyroid at birth or previous thyroid surgery.

Hashimoto’s thyroiditis or chronic lymphocytic thyroiditis is an autoimmune disease in which the thyroid gland is attacked by a variety of cell- and antibody-mediated immune processes. It was the first disease to be recognized as an autoimmune disease.It was first described by the Japanese specialist Hakaru Hashimoto in Germany in 1912.

There are many symptoms that are attributed to Hashimoto’s thyroiditis or Hashimoto’s disease. The most common symptoms include the following: fatigue, weight gain, pale or puffy face, feeling cold, joint and muscle pain, constipation, dry and thinning hair, heavy menstrual flow or irregular periods, depression, a slowed heart rate, and problems getting pregnant and maintaining pregnancy.

Hashimoto’s disease is about seven times more common in women than in men. It can occur in teens and young women, but more commonly shows up in middle age, particularly for men. People who get Hashimoto’s disease often have family members who have thyroid or other autoimmune diseases, and sometimes have other autoimmune diseases themselves.

The thyroid gland may become firm, large, and lobulated in Hashimoto’s thyroiditis, but changes in the thyroid can also be nonpalpable.Enlargement of the thyroid is due to lymphocytic infiltration and fibrosis rather than tissue hypertrophy. Physiologically, antibodies against thyroid peroxidase (TPO) and/or thyroglobulin cause gradual destruction of follicles in the thyroid gland. Accordingly, the disease can be detected clinically by looking for these antibodies in the blood. It is also characterized by invasion of the thyroid tissue by leukocytes, mainly T-lymphocytes. A rare but serious complication is thyroid lymphoma, generally the B-cell type, non-Hodgkin lymphoma.

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